There have been too few
studies to find etiologic associations, recent research is
pointing towards neurobiological involvement.
Reports by Wing (1981) of increased pre-, peri- or postnatal
complications were challanged by Szatmari et al (1989), who
found that complications during pregnancy or the postnatal
period were about the same in the control group.
Evidence from the family trees of there respective case loads
prompted Asperger, Wing as well as Szatmari to considered
the syndrome as genetically related. There seems to be a higher prevalence
of similar characteristics in the father.
Gillberg (1921) found that 5 of the 21 boys had prolonged brain
stem transmission time on auditory brainstem response
examination. 6 of the 21 had abnormal EEGs in the waking state
Gillberg noticed that 3 out of 18 children with Asperger he
studied, had slight or moderate atrophy of the brain.
Findings | Differential
Diagnosis | Treatment
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by Dr. Manaan Kar Ray